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Cardiac myopathy-induced by genetic alteration in abnormal myosin production Mya

ID: 95758 • Letter: C

Question

Cardiac myopathy-induced by genetic alteration in abnormal myosin production Myasthenia gravis-antibodies bind to and attack the body's own proteins Osteogenesis imperfecta-weak or fragile bones caused by a genetic defect in collagen production Congenital ectodermal dysplasia-defect in desmosomes causing skin that easily separates from deeper layers Phenylketonuria-genetic mutation in an enzyme that catabolizes the amino acid phenylalanine Tetrodotoxin poisoning from a parrotfish exposure-paralyzes muscle due to binding of fast sodium channels Type II diabetes mellitus-high blood glucose levels caused by decreased sensitivity to the hormone insulin

Explanation / Answer

Structure - Osteogenesis imperfecta - weak or fragile bones caused by genetic defect in collagen production

Cell Adhesion - Congenital ectodermal dysplasia - defects in chromosomes causing skin that easily separates from deeper layers.

Communication- Phenylketonuria - genetic mutation in an enzyme that catabolizes the amino acid phenylalanine

Membrane transport - Tetrodotoxin poison from a parrotfish exposure- paralysis muscle due to binding of fast sodium channels.

Catalysis - Cardiac myopathy - induced by genetic alteration in abnormal myosin production

Recognition - Myasthenia gravis - antibodies bind to and attack the body's own antigen.

Movement - Type II diabetes mellites - high blood glucose levels caused by decreased sensitivity to the hormone insulin.